Astiz Lab

Laboratory of Circadian Physiology | Achucarro Basque Center for Neuroscience

Sub-chronic exposure to the insecticide dimethoate induces a proinflammatory status and enhances the neuroinflammatory response to bacterial lypopolysaccharide in the hippocampus and striatum of male mice.


Journal article


M. Astiz, Y. Diz-Chaves, L. Garcia-Segura
Toxicology and Applied Pharmacology, 2013

Semantic Scholar DOI PubMed
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APA   Click to copy
Astiz, M., Diz-Chaves, Y., & Garcia-Segura, L. (2013). Sub-chronic exposure to the insecticide dimethoate induces a proinflammatory status and enhances the neuroinflammatory response to bacterial lypopolysaccharide in the hippocampus and striatum of male mice. Toxicology and Applied Pharmacology.


Chicago/Turabian   Click to copy
Astiz, M., Y. Diz-Chaves, and L. Garcia-Segura. “Sub-Chronic Exposure to the Insecticide Dimethoate Induces a Proinflammatory Status and Enhances the Neuroinflammatory Response to Bacterial Lypopolysaccharide in the Hippocampus and Striatum of Male Mice.” Toxicology and Applied Pharmacology (2013).


MLA   Click to copy
Astiz, M., et al. “Sub-Chronic Exposure to the Insecticide Dimethoate Induces a Proinflammatory Status and Enhances the Neuroinflammatory Response to Bacterial Lypopolysaccharide in the Hippocampus and Striatum of Male Mice.” Toxicology and Applied Pharmacology, 2013.


BibTeX   Click to copy

@article{m2013a,
  title = {Sub-chronic exposure to the insecticide dimethoate induces a proinflammatory status and enhances the neuroinflammatory response to bacterial lypopolysaccharide in the hippocampus and striatum of male mice.},
  year = {2013},
  journal = {Toxicology and Applied Pharmacology},
  author = {Astiz, M. and Diz-Chaves, Y. and Garcia-Segura, L.}
}





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